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A new discovery highlighted a novel purpose for intracellular MIF in regulating the accumulation of misfolded Cu/Zn superoxide dismutase (SOD1). Mutations in SOD1 are related to twenty% from the circumstances of familial amyotrophic lateral sclerosis, that's characterised by lack of motor neurons [72]. Neurodegenerative Issues, which includes Park
The amino-terminal proline residue is essential for the catalytic exercise; even so, it can be unclear irrespective of whether a functional enzyme activity of MIF is necessary for its biological function33,34,35,36. It is achievable that the enzyme actions of MIF signify vestigial signatures of the prevalent ancestral origin of MIF and DDT genes. s